Left anterior fascicular block
Encyclopedia
Left anterior fascicular block is a cardiac condition, distinguished from left bundle branch block
Left bundle branch block
Left bundle branch block is a cardiac conduction abnormality seen on the electrocardiogram . In this condition, activation of the left ventricle is delayed, which results in the left ventricle contracting later than the right ventricle....

.

It is caused by only the anterior half of the bundle of His
Bundle of His
The bundle of His, known as the AV bundle or atrioventricular bundle, is a collection of heart muscle cells specialized for electrical conduction that transmits the electrical impulses from the AV node to the point of the apex of the fascicular branches...

 being defective. It is manifest on the ECG
Electrocardiogram
Electrocardiography is a transthoracic interpretation of the electrical activity of the heart over a period of time, as detected by electrodes attached to the outer surface of the skin and recorded by a device external to the body...

 by left axis deviation.

It is much more common than left posterior fascicular block
Left posterior fascicular block
A left posterior fascicular block is a condition where the left posterior fascicle, i.e. the backside part of the left cardiac bundle, does not conduct the electrical impulses from the atrioventricular node...

.

Characteristics

Normal activation of the left ventricle proceeds down the left bundle branch, which consist of two fascicles the left anterior fascicle and left posterior fascicle. Left Anterior Fascicular Block (LAFB), which is also known as Left Anterior Hemiblock (LAHB), occurs when a cardiac impulse spreads first through the left posterior fascicle, causing a delay in activation of the anterior and lateral walls of the left ventricle which are normally activated via the left anterior fascicle.

Although there is a delay or block in activation of the left anterior fascicle there is still preservation of initial left to right septal activation as well as preservation of the inferior activation of the left ventricle (preservation of septal Q waves in I and aVL and small initial R wave in leads II, III, and aVF). The delayed and unopposed activation of the remainder of the left ventricle now results in a shift in the QRS axis leftward and superiorly, causing marked left axis deviation. This delayed activation also results in a widening of the QRS complex, although not to the extent of a complete LBBB

Criteria

  • Abnormal left axis deviation (usually between –45° and –60°)
  • qR complex in the lateral limb leads (I and aVL)
  • rS pattern in the inferior leads (II, III, and aVF)
  • Delayed intrinsicoid deflection
    Intrinsicoid deflection
    In the electrocardiogram, the time of the onset of the intrinsicoid deflection, also referred to as the R wave peak time, is measured from the beginning of the QRS complex to the peak of the R wave...

    in lead aVL (> 0.045 s)


It is important not to call LAFB in the setting of a prior inferior wall myocardial infarction which may also demonstrate left axis deviation due to theinitial forces (Q wave in a Qr complex) in leads II, III, and aVF. As opposed to LAHB, the left axis shift is due to terminal forces (i.e., the S wave in an rS complex) being directed superiorly,

Effects of LAFB on Diagnosing Infarctions and Left Ventricular Hypertrophy

LAHB may be a cause of poor R wave progression across the precordium causing a pseudoinfarction pattern mimicking an anteroseptal infarction. It also makes the electrocardiographic diagnosis of LVH more complicated, because both may cause a large R wave in lead aVL. Therefore to call LVH on an EKG in the setting of an LAHB you should see the presence of a “strain” pattern when you are relying on limb lead criteria to diagnose LVH.

Clinical Significance

  • It can be seen in approximately 4% of cases of acute myocardial infarction
    • It is the most common type of intraventricular conduction defect seen in acute anterior myocardial infarction, and the left anterior descending artery is usually the culprit vessel.
    • It can be seen with acute inferior wall myocardial infarction.
  • It also associated with hypertensive heart disease, aortic valvular disease, cardiomyopathies, and degenerative fibrotic disease of the cardiac skeleton.

External links

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